🎾 Extracorporeal Shockwave Therapy Simulator
A model of extracorporeal shockwave therapy to stimulate healing in chronic tendinopathy of the extensor carpi radialis brevis.
Chronic Lateral Epicondyle Tendinopathy
Repeated strain outpaces repair at the tendon origin.
- 1–3%: Condition prevalence (of adults yearly)
- 35–54: Peak age range (years old)
- 6+ mo: Typical symptom duration (before treatment)
- Angiofibroblastic: Tissue pattern (degeneration, not inflammation)
A failed healing response
Microtears accumulate faster than the tendon can repair them.
Poor local blood supply
Sparse vessels starve the injured origin of nutrients.
Disorganized collagen
Fibers lose their normal parallel, load-bearing alignment.
Chronic tendinopathy is degenerative, not simply inflamed.
Focused Extracorporeal Shockwave Application
An external applicator fires focused acoustic pulses at the tendon.
- 1,500–2,500: Pulses per session (typical dose)
- 0.08–0.6: Energy flux density (mJ/mm²)
- Weekly: Session frequency (for 3–5 sessions)
- Millimeter: Focal depth control (precision targeting)
Focused acoustic energy
Sound waves converge precisely on the tendon origin.
Energy level matters
Higher energy reaches deeper but needs more tolerance.
Non-invasive delivery
No incision is needed to reach the injured tissue.
Energy dose is tuned to balance effect and comfort.
Microtrauma Triggers a Renewed Healing Cascade
Tiny controlled injuries restart the body's repair machinery.
- Micro: Cavitation bubbles (formed at impact zone)
- TGF-β, VEGF: Growth factor release (upregulated locally)
- 2–6 wk: Pain relief onset (after first sessions)
- Fibroblast: Cellular response (activation begins)
Deliberate microdamage
Small controlled tears reawaken a stalled repair signal.
Growth factor surge
Local cells release factors that summon repair machinery.
Pain gate modulation
Nerve signaling shifts, easing pain within weeks.
Reinjury paradoxically restarts genuine healing.
Neovascularization Restores Blood Supply
New capillaries sprout into the tendon, carrying nutrients in.
- Weeks 2–8: New vessel growth (after treatment starts)
- Up to 3×: Vessel density gain (versus baseline)
- VEGF: Key vascular driver (signaling pathway)
- Improved: Oxygen delivery (tissue perfusion)
Capillary sprouting
New microvessels branch inward from healthy tissue edges.
Sessions drive vessel growth
More completed sessions yield denser vascular networks.
Nutrient delivery restored
Fresh blood flow finally reaches the injured core.
Vessel density tracks closely with sessions completed.
Tissue Remodeling and Collagen Reorganization
Over weeks, fibers realign into strong, organized tendon tissue.
- 6–12 wk: Remodeling timeline (for full effect)
- 65–85%: Clinical success rate (pain improvement)
- III → I: Collagen type shift (toward mature fiber)
- Low: Recurrence risk (with completed course)
Collagen realignment
Fibers reorganize along the tendon's natural load lines.
Strength returns gradually
Tensile strength rebuilds over weeks, not days.
Durable outcome
Improved vascularity keeps healing from stalling again.
Full remodeling can take up to three months.
A model of extracorporeal shockwave therapy to stimulate healing in chronic tendinopathy of the extensor carpi radialis brevis.
2D · HTML5 Canvas 2D · 60 FPS target · runs fully client-side, no install