💅 Ringworm (Tinea Corporis) Contagion & Treatment Simulator
This simulation models the spread of a dermatophyte infection on the body through contact, demonstrating the characteristic ring-like rash and the effectiveness of topical antifungal treatments.
Contact With An Infected Source
Ringworm spreads through direct contact with contaminated skin, fur, or surfaces.
- High: Person-to-person risk (skin-to-skin contact)
- Moderate: Zoonotic risk (pets, livestock)
- Weeks: Fomite survival (on surfaces)
- 4–14 d: Incubation period (before rash appears)
Human contact
Wrestling, shared bedding, and skin contact spread spores between people.
Animal contact
Cats, dogs, and livestock often carry dermatophytes without visible symptoms.
Surface contact
Gym mats, towels, and combs harbor infectious spores for weeks.
Spores Land On New Skin
Microscopic fungal spores attach to the outer keratin layer of fresh skin.
- 2–5 µm: Spore size (microscopic arthrospores)
- Minutes: Adherence time (to attach to keratin)
- Warm/moist: Ideal conditions (sweat, occlusion)
- Yes: Minor trauma needed (micro-abrasions help entry)
Keratin affinity
Dermatophytes uniquely digest keratin, the protein of skin, hair, and nails.
Moisture and friction
Sweaty, occluded skin areas favor spore germination and hyphal growth.
Barrier breach
Tiny scratches or abrasions let spores penetrate the stratum corneum.
The Ring-Shaped Rash Forms
Fungal hyphae grow radially outward, creating the characteristic annular lesion.
- Centrifugal: Growth pattern (outward from origin)
- 1–5 cm: Ring diameter (typical untreated size)
- Common: Central clearing (active border, clear center)
- Mild–mod: Itch severity (pruritic border)
Active border
The advancing edge is scaly, red, and where live fungus concentrates.
Central clearing
Older central skin heals as the fungus advances outward only.
Multiple lesions
Repeated contact can seed several rings across the body.
Antifungal Cream Application
Daily topical antifungals halt fungal growth at the active rash border.
- Azoles: First-line agents (clotrimazole, terbinafine)
- 1–2×/day: Application freq (beyond rash edge)
- 2–4 wk: Typical course (topical therapy)
- >80%: Cure rate (uncomplicated cases)
Mechanism
Azoles and allylamines disrupt fungal cell membrane synthesis directly.
Application area
Cream must extend past the visible border to catch spreading hyphae.
Adherence matters
Stopping early after symptoms fade risks relapse and regrowth.
Rash Resolves Over Weeks
Consistent treatment shrinks the ring until skin returns to normal.
- 1–2 wk: Visible improvement (reduced redness)
- 4–6 wk: Full clearance (typical timeline)
- Low: Recurrence risk (if course completed)
- Drops fast: Contagion after Rx (within days of therapy)
Shrinking ring
The active border retreats inward week by week under treatment.
Reduced contagion
Treated lesions shed far fewer viable spores to others.
Preventing recurrence
Treating pets, disinfecting items, and finishing the course prevent relapse.
This simulation models the spread of a dermatophyte infection on the body through contact, demonstrating the characteristic ring-like rash and the effectiveness of topical antifungal treatments.
2D · HTML5 Canvas 2D · 60 FPS target · runs fully client-side, no install