Anaphylaxis Trigger ID

Matching clinical pattern and exposure timing to the likely anaphylaxis trigger category.

Current Stage

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Category likelihood

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Onset speed

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Diagnostic confidence

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Avoidance advice

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Trigger CategoryFood

Exposure → Onset (min)5

Food allergen trigger

Insect sting venom trigger

Medication (drug) trigger

Latex contact trigger

Reaction onset marker

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Food-Triggered Anaphylaxis — Recognizing the Pattern

Food allergens are a leading cause of anaphylaxis worldwide.

Peanut, tree nut: Common foods (Top pediatric triggers)
10–60 min: Typical onset (After ingestion)
Oral / ingestion: Route (GI absorption first)
GI + skin + resp: Key sign (Multi-system pattern)

Clinical clues for food triggers

Short placeholder: symptoms follow a meal, GI signs prominent, delayed onset compared to venom.

Insect Sting Venom — Rapid Systemic Reaction

Hymenoptera stings cause some of the fastest anaphylaxis onsets.

Bee, wasp, ant: Common insects (Venom-injecting stings)
1–15 min: Typical onset (After sting)
Intradermal venom: Route (Direct systemic entry)
Rapid cardiovascular drop: Key sign (Fast progression)

Clinical clues for venom triggers

Short placeholder: sting site visible, very fast onset, hypotension risk elevated.

Drug-Induced Anaphylaxis — Variable Kinetics

Medications trigger anaphylaxis via IV, oral, or topical routes.

Antibiotics, NSAIDs: Common drugs (Frequent culprits)
1–30 min: Typical onset (Route-dependent)
IV/IM/oral: Route (Fastest via IV)
Temporal drug link: Key sign (Recent administration)

Clinical clues for drug triggers

Short placeholder: onset tied closely to dose timing, route changes speed.

Latex-Triggered Reaction — Contact & Mucosal Exposure

Latex exposure via gloves or devices can provoke anaphylaxis.

Gloves, catheters: Common sources (Healthcare settings)
15–60 min: Typical onset (Contact-dependent)
Skin/mucosal contact: Route (Slower absorption)
Occupational history: Key sign (Repeated exposure risk)

Clinical clues for latex triggers

Short placeholder: healthcare or occupational exposure context, contact dermatitis history.

Integrating Exposure Timeline with Clinical Presentation

Combining onset timing and exposure history narrows the trigger.

Time-to-onset: Key input (Minutes since exposure)
Exposure route: Key input (Oral, sting, drug, contact)
Likely category: Output (Ranked by pattern fit)
Avoidance plan: Output (Trigger-specific advice)

Diagnostic synthesis approach

Short placeholder: match onset window and route to each candidate category.

Short placeholder: confirmatory testing follows clinical identification.